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November 21, 2011Faculty Publication: Drevets et al.
November 9, 2011Meyers, N., Fromm, S., Luckenbaugh, D. A., Drevets, W. C., & Hasler, G. (2011). Neural correlates of sleepiness induced by catecholamine depletion. Psychiatry Research: Neuroimaging, 194(1), 73-78. doi: 10.1016/j.pscychresns.2011.06.015
Although extensive indirect evidence exists to suggest that the central dopaminergic system plays a significant role in the modulation of arousal, the functional effect of the dopaminergic influence on the regulation of the sleep–wake cycle remains unclear. Thirteen healthy volunteers and 15 unmedicated subjects with a history of major depressive disorder underwent catecholamine depletion (CD) using oral alpha-methyl-para-tyrosine in a randomized, placebo-controlled, double-blind, crossover study. The main outcome measures in both sessions were sleepiness (Stanford-Sleepiness-Scale), cerebral glucose metabolism (positron emission tomography), and serum prolactin concentration. CD consistently induced clinically relevant sleepiness in both groups. The CD-induced prolactin increase significantly correlated with CD-induced sleepiness but not with CD-induced mood and anxiety symptoms. CD-induced sleepiness correlated with CD-induced increases in metabolism in the medial and orbital frontal cortex, bilateral superior temporal cortex, left insula, cingulate motor area and in the vicinity of the periaqueductal gray. This study suggests that the association between dopamine depletion and sleepiness is independent of the brain reward system and the risk for depression. The visceromotor system, the cingulate motor area, the periaqueductal gray and the caudal hypothalamus may mediate the impact of the dopaminergic system on regulation of wakefulness and sleep. (PsycINFO Database Record (c) 2011 APA, all rights reserved) (journal abstract)
Link to article record in Schusterman Library catalog, and possible full-text
You too can participate in OU-Tulsa Reads
November 8, 2011January 17 – March 31, 2012 at the OU-Tulsa Schusterman Center Library Gallery
People who can say of a book, “This has changed my life” truly understand the meaning of happiness ~ Daisaku Ikeda
In January, 2012, the OU-Tulsa Schusterman Library will host OU-Tulsa Reads, a multimedia exhibit about books that have had a powerful impact on community members. Administrators, faculty, staff, and students are invited to share the books that have inspired them and served as catalysts for personal, professional, or educational changes in their lives. Through sharing our most inspiring reads, we will share the powerful impact of reading as well as an important part of who we are as a community.
What book has inspired you? Include your book in our exhibit!
Faculty publication: Ma, Cheng
November 7, 2011Ma, R., & Cheng, S. (2011). The universality of generalized hamming code for multiple sources. [Article]. IEEE Transactions on Communications, 59(10), 2641-2647. doi: 10.1109/tcomm.2011.081711.100211
We consider zero-error Slepian-Wolf coding for a special kind of correlated sources known as Hamming sources. Moreover, we focus on the design of codes with minimum redundancy (i.e., perfect codes). As shown in a prior work by Koulgi et al., the design of a perfect code for a general source is very difficult and in fact is NP-hard. In our recent work, we introduce a subset of perfect codes for Hamming sources known as Hamming Codes for Multiple Sources (HCMSs). In this work, we extend HCMSs to generalized HCMSs, which can be proved to include all perfect codes for Hamming sources. To prove our main result, we first show that any perfect code for a Hamming source with two terminals is equivalent to a Hamming code for asymmetric Slepian Wolf coding (c.f. Lemma 2). We then show that any multi-terminal (of more than two terminals) perfect code can be transformed to a perfect code for two terminals (c.f. Lemma 3) and to a perfect code with an asymmetric form (c.f. Lemma 4). Equipped with these results, we prove that every perfect Slepian-Wolf code for Hamming sources is equivalent to a generalized HCMS. [ABSTRACT FROM PUBLISHER]
Faculty Publication: Chalmers, Copeland, Hester, Fields, Gardner
October 31, 2011Chalmers, L. J., Copeland, K. C., Hester, C. N., Fields, D. A., & Gardner, A. W. (2011). Paradoxical increase in arterial compliance in obese pubertal children. [Article]. Angiology, 62(7), 565-570. doi: 10.1177/0003319711399117
We determined whether arterial compliance measured by pulse wave analysis is impaired in obese pubertal children compared to normal weight controls, and assessed whether arterial compliance is associated with ambulatory activity. Body fat percentage was significantly different between the normal (n = 33) and obese (n = 34) participants (P < .001). Large (P = .012) and small (P < .001) arterial compliance were lower in the normal-weight group. After adjusting for height, systolic and diastolic blood pressure, race, sex, and Tanner stage, large arterial compliance was no longer different between groups (P = .066), whereas small arterial compliance remained higher in the obese group (P < .001). Obese pubertal children have paradoxically increased small arterial compliance compared to that of normal weight children, even after adjusting for height, blood pressure, race, sex, and Tanner stage. Thus, obesity in adolescence is not associated with impairments in small arterial compliance. [ABSTRACT FROM PUBLISHER]
Link to article record in Schusterman Library catalog and possible full text
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